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Psychedelics in Multiple Sclerosis: Mechanisms, Challenges, and Prospects for Neuroimmune Modulation and Repair

Specifically, AA is converted to prostaglandins via the cyclooxygenase pathway, to leukotrienes and lipoproteins via the lipoxygenase pathway, and to epoxyeicosatrienoic acid and hydroxyeicosatetraenoic acid via the cytochrome P450 pathway (58)

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We have already discussed that released GDNF can inhibit microglial activation in vivo and in vitro , thereby revealing a possible interaction between these glial cells in modifying (microglial-mediated) neuroinflammation (Rocha et al., 2012