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demonstrated that the proportion of μ-opioid receptor–positive B cells was lower than that in controls in blood samples from patients with FMS, suggesting that B cells μ-opioid receptor deficiency in B cells may impair endogenous opioid analgesic activity and contribute to sensitization, thereby supporting a pathological mechanism support for the aggravation of FMS and may prove the role for B-cell subsets in the pathogenesis of NPP (102)

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