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Moreover, BNIP3 and BNIP3L compete with Beclin-1 to bind to BCL2 and BCL-XL through their intrinsic BH3 atypical domain, resulting in disruption of the BCL2/BCL-XL-Beclin-1 complex and release of Beclin-1, which is required for autophagic activity [57]
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[DOI] [PubMed] [Google Scholar] 98.Kalantar-Zadeh K., Block G., Humphreys M.H., McAllister C.J., Kopple J.D
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