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In addition, synaptic copper dysregulation impairs hippocampal function via multiple pathways, including competitive inhibition of NMDA-R at glutamatergic synapses, suppression of the cAMP response element-binding protein (CREB) pathway via PP1/PP2A phosphatase activation, and impaired activity-dependent brain-derived neurotrophic factor (BDNF) secretion, which is vital for synaptic plasticity and memory formation (14)
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The basal GSH level in astrocytes was greater than that in neurons
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