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In vivo , intravenous NR-LGNPs (10 mg/kg) attenuate myocardial injury by reducing CK-MB, LDH, AST, and ALT, restoring antioxidants (SOD, CAT, GSH), decreasing LPO (MDA, TBARS), improving lipid profiles, normalizing systolic blood pressure, and correcting electrocardiographic abnormalities, while histopathology and 2,3,5-triphenyltetrazolium chloride (TTC) staining confirm preserved myocardial architecture and reduced infarct size

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It is crucial to maintain a healthy redox state of pancreatic β-cells, as their ability to secrete insulin in response to glucose is dependent on intracellular thiols [30]