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This unifying concept in vitreo-retinopathies is based upon the premise that same initiating abnormality (i.e., excess liquefaction without concurrent vitreo-retinal dehiscence) can explain several seemingly disparate vitreo-retinal disorders ranging from retinal tears/detachments to axial vitreo-macular traction syndrome and tangential vitreo-maculopathies such as macular pucker and macular holes

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Compared with WT SOD1, disease-linked mutant SOD1 proteins readily form monomers by reduction of the disulfide bond between Cys-57 and Cys-146 or by demetallation at the dimer interface (Tiwari and Hayward, 2003