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Pain Processing Mechanisms Central Sensitization ✓ (via mGlu2 receptor upregulation)[4][5] Neuroinflammation ✓ (modulates glial activation)[12][13] Mitochondrial Dysfunction ✓ (enhances ATP production, β-oxidation)[2][13] Oxidative Stress ✓ (direct antioxidant activity)[2][13] Systemic Inflammation – limited direct evidence — SUMMARY OVERVIEW CLINICAL PEARLS 1
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