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Experimental studies have shown that antioxidant compounds can improve follicular cell survival, support mitochondrial function, and reduce inflammatory signaling pathways associated with hair loss.¹ Although further clinical research is needed, improving antioxidant support may represent a promising strategy for preserving follicle vitality
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The classical M1 phenotype is associated with the release of pro-inflammatory molecules and activation of receptors, and the M2 phenotype is related to the secretion of anti-inflammatory mediators and growth factors
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