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In contrast, the development of hepatic steatosis and apparent disruption of the hepatic metabolome are characteristic of B6/N mice fed STD under TN conditions, which may potentially serve as a model for investigating early pathophysiological changes associated with the development and progression of NAFLD
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Ischemia–reperfusion notably increased oxidative stress, apoptosis, and inflammatory markers, as shown by higher levels of malondialdehyde, caspase-3, TNF-α, and IL-6 ( p 0.05, and homogeneity of variance using Bartlett’s test before the ANOVA test